INTRODUCTION
People with end-stage kidney disease (ESKD) who are undergoing
haemodialysis commonly experience disturbances in serum
potassium (K) (Kovesdy et al. 2007). In particular, patients are at
high risk of hyperkalaemia as their chronic renal disease
progresses, primarily due to inefficient renal K excretion
(Zehnder et al. 2001; Kovesdy et al. 2007; Al-Ghamdi et al.
2010). Hyperkalaemia is associated with increased risk of
mortality for patients on dialysis (Kovesdy et al. 2007), including
sudden cardiac arrest (Pun et al. 2011). Given that cardiac
disease is the leading cause of death for patients with ESKD
who are on dialysis (Santoro et al. 2008; Pun et al. 2011),
effective management of K removal during dialysis is critically
important.