and some PASpositive substance in consequence of pronounced haemorrhages.
Often tissue elements surrounding the tracks were affected by a pronounced
coagulative necrosis (Fig. 4). The adjoining portal areas and
the congested sinusoids were abundantly infiltrated by eosinophils,
lymphocytes, and macrophages (Fig. 5). Some irregular fragments
from mesenchymal tissue often occurred in the larger tracks resulting
from extensive damage. The fine-grained brown material in the peripherial
areas of some tracks was similar to the residue of ingested
substance in the caeca of immature flukes and apparently had been
defaecated by them. An incomplete rim of degenerated hepatocytes
surrounding such tracks appeared more dark-stained compared to
other areas (Fig. 6).
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