Accurate definition and better understanding of the mechanisms of stroke are crucial as this will guide the effective care and therapy.
In this paper, we review the previous basic and clinical researches on the causes ormechanisms of ischemic cerebrovascular diseases
(ICVD) and interpret the correlation between embolism and hypoperfusion based on vascular stenosis and arterial intimal lesions.
It was suggested that if there is no embolus (dynamic or in situ emboli), there might be no cerebral infarction. Three kinds of
different clinical outcomes of TIA were theoretically interpreted based on its mechanisms. We suppose that there is a correlation
between embolism and hypoperfusion, and which mechanisms (hypoperfusion or hypoperfusion induced microemboli) playing
the dominant role in each type of ICVD depends on the unique background of arterial intimal lesions (the vulnerability of vessels).
That is to say, the vulnerability of vessels is involved in the role of embolism and hypoperfusion in the mechanisms of ischemic
cerebrovascular diseases.This inference might enrich and provide better understandings for the underlying etiologies of ischemic
cerebrovascular events.