It
appears that ABA increases tissue sensitivity to ET, affecting
seed germination [39,55], whereas ET may suppress seed
dormancy by inhibiting ABA action [46,55]. In recent studies
using ET response mutants of Arabidopsis, endogenous ET
promoted seed germination by decreasing sensitivity to
endogenous ABA [55]. ET appears to be a negative regulator
of ABA during germination [39,55] (Fig. 4). In short, ET seems
to act antagonistically with ABA during dormancy termination
and acts in concert with GAs to promote these transitional
changes. Although ET and GAs work together in the process of
radicle emergence, the participation of GAs appears to be
quantitatively and qualitatively more importan
Itappears that ABA increases tissue sensitivity to ET, affectingseed germination [39,55], whereas ET may suppress seeddormancy by inhibiting ABA action [46,55]. In recent studiesusing ET response mutants of Arabidopsis, endogenous ETpromoted seed germination by decreasing sensitivity toendogenous ABA [55]. ET appears to be a negative regulatorof ABA during germination [39,55] (Fig. 4). In short, ET seemsto act antagonistically with ABA during dormancy terminationand acts in concert with GAs to promote these transitionalchanges. Although ET and GAs work together in the process ofradicle emergence, the participation of GAs appears to bequantitatively and qualitatively more importan
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