Secondary hyperparathyroidism in vitamin D receptor-null mice may also contribute to renin upregulation [19], considering that intravenous infusion of PTH increases plasma renin activity and renin release [23, 24].
Secondaryhyperparathyroidism in vitamin D receptor-null mice may also contribute to renin upregulation [19], considering that intravenous infusion of PTH increases plasma renin activity and renin release [23, 24].