have previously reported in these pigs (Pearce et al., 2013). We previously reported that HS pigs had a marked and sustained increase in rectal temperatures (1.5°C) and respiration rates (two-fold) compared to TN and PFTN pigs over the 7-d test period. Furthermore, HS pigs consumed less feed starting from d 1 of HS and remained constantly reduced over the 7 d period (46%) compared to TN pigs. By design, the PFTN pigs feed intake mirrored that of the HS pigs. Pigs in TN conditions gained body weight (1.14 kg/d) throughout the experiment, while the HS pigs initially lost 2.7 kg of BW at d 1, but gained 0.03 and 1.65 kg cumulatively by d 3 and 7, respectively compared to initial body weights. The PFTN pigs had lost 2.47 kg of BW by d 7. These phenotypic changes may be related to a decrease in intestinal integrity and an increase in endotoxin permeability. Interestingly, our data suggests that feed intake restriction may orchestrate some of this stress related response as explained by our PFTN treatment.