The classical forms of allergic
bronchopulmonary mycosis include: increased
severity of asthma, transient infiltrates in the lungs,
large amount of sputum production, increased total
IgE, immediate hypersensitivity reactions to
Aspergillus spp, and eosinophilia (Agarwal et al.,
2011; Khosravi et al., 2007). Thioredoxin is an
allergenic protein from the mould Aspergillus
fumigatus, the aetiologic agent identified in the
majority of Aspergillus-related asthma, and from
Malassezia sympodialis, a skin-colonizing yeast
involved in the pathophysiology of asthma and
atopic eczema. Spores of Malassezia could be a
trigger of asthma in atopic people (Limacher et al.,
2007).