Among the RTE products in this study, only chicken skin and breast meat contained all five HCAs. PhIP in chicken skin was very high, especially in replication 1 (27.27 ng/g total HCAs) (Table 3). This agrees with results of Liao, Wang, Xu, and Zhou (2010) who found that PhIP can form easily in cooked chicken. As mentioned previously, the skin of chicken replication 1 had more fat and protein and less moisture than other replications. Consequently, the total amount of HCAs in the skin of chicken replication 1 (41 ng/g) was much higher than that in the other replications (2 to 5 ng/g); PhIP and MeIQx were most abundant (Table 2). This result agrees with other research that showed an increase in HCA levels as moisture content decreased (Murkovic, 2004). The amount of HCAs in rotisserie chickens in the present study are in the same range as amounts reported by Knize et al. (1998), who showed 0.45 ng/g of MeIQx and 0.75 ng/g of PhIP in white chicken meat and 0.40 ng/g of MeIQx and 0.59 ng/g of PhIP in dark chicken meat; however, they did not report HCAs in chicken skin. In this study, total HCAs were 1.56 ng/g in rotisserie chicken meat and 13.08 ng/g in the skin. These findings indicate that HCA exposure can be reduced by not eating chicken skin.