precise pathogenesis of the disease is uncertain, there appears to be decreased laminar perfusion, with varying degrees of inflammation and necrosis of the sensitive laminae. Causes include carbohydrate overload, endotoxaemia (such as caused by gastrointestinal disease, septic metritis and pleuritis), excessive work, and hyperadrenocorticism (Cushing’s syndrome).
precise pathogenesis of the disease is uncertain, there appears to be decreased laminar perfusion, with varying degrees of inflammation and necrosis of the sensitive laminae. Causes include carbohydrate overload, endotoxaemia (such as caused by gastrointestinal disease, septic metritis and pleuritis), excessive work, and hyperadrenocorticism (Cushing’s syndrome).
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