The precise manner by which dietary NO3
− supplementation can affect aerobic efficiency at
sub-maximal workloads remains unclear, but several mechanisms have been postulated to explain this
phenomenon. In detail, a NO-mediated enhancement in mitochondrial efficiency and/or enhanced
muscle blood flow have been reported, even though some have argued that this effect could be related
to NO-mediated effect on muscle contractile function [2,4,6–8,17].
The precise manner by which dietary NO3− supplementation can affect aerobic efficiency atsub-maximal workloads remains unclear, but several mechanisms have been postulated to explain thisphenomenon. In detail, a NO-mediated enhancement in mitochondrial efficiency and/or enhancedmuscle blood flow have been reported, even though some have argued that this effect could be relatedto NO-mediated effect on muscle contractile function [2,4,6–8,17].
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