In the future, targeted inhibition of the morphological switch
from yeast-to-hyphal growth would be a very attractive
option for specifically controlling C. albicans infections, both
prophylactically and as a treatment. As discussed above, such
an inhibition could prevent adhesion, invasion and damage
of epithelial and endothelial cells, may prevent escape from
macrophages and from the bloodstream, and may reduce the iron
acquisition capacity of the fungus.