To summarize, the current paper evaluated the contribution
of verbal and associative demands in giving rise to
PAL deficits in dyslexia. Contrary to the predictions of the
crossmodal hypothesis, children with dyslexia exhibited
deficits specific to PAL tasks with a verbal output demand,
regardless of whether the mappings were crossmodal or
unimodal in nature. Additionally, PAL deficits were shown
to be a consequence of difficulties in phonological form
learning, rather than difficulties with associative learning.
Taken together, these results caution against interpreting
the reading and spelling difficulties of children with dyslexia
as consequences of poor or inefficient orthographyphonology
mappings. Although there is no doubt that children
with dyslexia demonstrate poor learning in tasks that
require such mappings (e.g., letter learning, reading), our
data suggest that these deficits are neither specific to
crossmodal mappings, nor do they arise at the level of
the association.
To summarize, the current paper evaluated the contributionof verbal and associative demands in giving rise toPAL deficits in dyslexia. Contrary to the predictions of thecrossmodal hypothesis, children with dyslexia exhibiteddeficits specific to PAL tasks with a verbal output demand,regardless of whether the mappings were crossmodal orunimodal in nature. Additionally, PAL deficits were shownto be a consequence of difficulties in phonological formlearning, rather than difficulties with associative learning.Taken together, these results caution against interpretingthe reading and spelling difficulties of children with dyslexiaas consequences of poor or inefficient orthographyphonologymappings. Although there is no doubt that childrenwith dyslexia demonstrate poor learning in tasks thatrequire such mappings (e.g., letter learning, reading), ourdata suggest that these deficits are neither specific tocrossmodal mappings, nor do they arise at the level ofthe association.
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