The literature report 3 theories in the etiology of mesiodens, but this subject remains controversial. (a) Phylogenic reversion or atavism[5] (b) Dichotomy theory[1] and (c) Hyperactivity of the dental lamina.[6] Autosomal dominant inheritance with incomplete penetration has been the proposed genetic theory. A sex-linked pattern has also been proposed as males are affected twice as frequently as females.[7] Mesiodens can be classified on the basis of their occurrence in the permanent dentition (rudimentary mesiodens) or the primary dentition (supplementary mesiodens) and according to their morphology (conical, tuberculate or molariform).[