1. Introduction
The exact mechanism of reduction of fertilizing ability
of cryopreserved spermatozoa is not yet completely
understood. However, there have been studies on the
role of reactive oxygen species (ROS) in the functioning
of spermatozoa and their fertilizing ability after
liquid storage and cryopreservation [1–5]. Many studies
have shown that the main reason for abnormal
sperm functioning is lipid peroxidation (LPO) and antioxidant
imbalance, caused by the occurrence of oxidative
stress [6–9].