Pacemaker displacement, depending on its location, leads to an increase in heart rate or uncoordinated contraction, interfering with effective filling and con-traction. As demonstrated by the literature, blocking the transmission of the cardiac impulse results in decreased heart rate and the occurrence of multiple episodes during which ventricular pumping stops. Furthermore, impulse transmission through abnormal pathways in the heart leads to uncoordinated contraction and distension of the chambers and significantly interferes with cardiac pumping. Finally, abnormal spontaneous impulses in the atrium as well as in the AV beam or node interfere with total filling of ventricles and lead to decreased or nearly zero systolic output (Guyton and Hall, 2006).