Candida dubliniensis was first described in 1995 from oral
cavities of human immunodeficiency virus (HIV)-infected individuals
[1]. The species forms only a minor component of normal
microbiota but has a worldwide distribution [2]. Despite its close
relationship with C. albicans, which is the predominant pathogenic
species, the etiopathologic role of C. dubliniensis has mostly been
restricted to oral candidiasis. In recent years, however, C.
dubliniensis has increasingly been reported from patients with
candidemia [3–11]. Although the species is significantly less
virulent and genetically more clonal than C. albicans [12–14], the
reasons for its expanding role in invasive disease remain largely
unknown. Here, we report the prevalence of C. dubliniensis in
various clinical specimens over a nine-year period and discuss its
role in nosocomial candidemia.