The molecular target of OP-induced acute toxicity is AChE, whose inhibition in synapses and neuromuscular junctions causes the accumulation of the neurotransmitter acetylchloine, inducing overstimulation of the nervous system
The molecular target of OP-induced acute toxicity isAChE, whose inhibition in synapses and neuromuscularjunctions causes the accumulation of the neurotransmitteracetylchloine, inducing overstimulation of the nervous system