The 179 genotypes with between one and three marker alleles for the CMD2 gene were phenotypically shown to be resistant to the disease. Since they resulted mainly from crosses between CMD2 donor parents (from seven CIAT lines) and landraces (with
TMEII and Dabodabo being CMD resistant), resistance in these genotypes may result from the CMD2 source alone or from both the CMD2 source and any other new source of CMD resistance if we assume that the CMD resistant landrace progenitors (Dabodabo and TMEII) represent a new source of CMD resistance genes. Rabbi et al. (2014) used the genetic map to anchor the seven molecular markers previously reported to be linked to the dominant gene resistance. They found all the previously related markers (except
for RME1 and RME4) to be located on scaffolds occurring in the same region of linkage group 16,indicating that three of the four markers used in this study were located in this region. The single locus on linkage group 16 which explained 74 % of phenotypic variation was co-located with the SSRY28 marker. They found a total of 281 SNPs at this locus