The cytokine and chemokine milieu that is generated during these innate responses drives the preferential differentiation of IL-17-producing Th17, IFN-γ-producing Th1, and IL-17 or IFN-γ-co-producing Th1 or Th17 cells,124 facilitates the recruitment of neutrophils to the intestinal lamina propria, and consequently restrict the overgrowth of gut fungi.