suppressed cytokine production and iNOS expression, while the more polar fractions (BuOH and H2O extracts) were found to selectively block COX-2 activation. Also, different patterns were similarly obtained in an antioxidant activity assay in which EtOAc was higher in the XO activity assay system ,but BuOH in DPPH assay, although exact pharmacological mechanism is not fully understood in this study. Moreover, Fig. 2 proposes that there may be some principles blocking the post-translational steps of the inflammatory gene products, as in the case of some natural products and chloroquine which inhibit matalloproteinase, which is involved in cytokine processing (Jeong andJue, 1997). Therefore, our data indicate that Cinnamomum camphora may possess two different types of anti-oxidative and anti-inflammatory compounds, which have distinguishable target enzyme(s). Furthermore, this pharmacological property of Cinnamomum camphora may be advantageousin terms of its usage in various attacking inflammatory diseases, considering that a number of cellular inflammatory phenomena are or chestrated in the onset of inflammatory diseases