Zika virus (ZIKV), a member of the vector-borne Flaviviridae family, has emerged as a new public health threat due to a series of recent outbreaks and links to systemic neurological pathologies [1]. Symptoms of ZIKV infections include fever, rash, conjunctivitis and arthralgia, which can often lead to misdiagnosis as the more common dengue virus (DENV) infection [2]. The spread of ZIKV now has become a critical and urgent issue because the 2015 ZIKV outbreak in northern Brazil coincided with a spike in the incidence of microcephaly in newborns [3]. The infection was also linked to systemic neurological disorders and morphological alterations of neuronal organs [1]. ZIKV has been identified in brain tissue and amniotic fluid from microcephaly cases [4] and [5]. ZIKV neurotropism and direct causation were shown both at the cellular level and in an animal model [6], [7] and [8]..