Septic shock is the primary cause of death in critical care unitsbecause of tissue hypoperfusion and multiorgan failure.Infection and mediators induced by infection result in hypo-volemia, vascular failure, and heart failure [1,2].Four physiologic subtypes of shock are known as follows:hypovolemic, cardiogenic, distributive, and obstructive shock.Septic shock was recently classified, according to patho-physiological background, into two stages as follows: the earlyhypovolemic and the late vascular and myocardial circulatorydysfunction stage [3].Although warm shock, mediated by endotoxin-inducedabnormal vasodilation, is a major characterization fordistributive type septic shock, cold cardiogenic shock duringsepsis, resulting from cardiac poor perfusion during septicshock or underlying cause of sepsis, is associated with elevatedtroponin and B-type natriuretic peptide levels (cardiacdysfunction markers) and diminished cardiac function diag-nosed by echocardiography [4]. During hypovolemic shock