In our previous study, the transient middle cerebral artery occlusion (tMCAO) model was used to investigate the e↵ect of PTH on cerebral ischemia/reperfusion injury. The results showed that PTH significantly improves neurological deficit infarct volume, and downregulates the levels of IL-1, IL-6, and TNF brain tissue of tMCAO rats. Furthermore, PTH decreases the cytosolic levels of cytochrome