Abstract
Parathyroid hormone (PTH), vitamin D, and magnesium interact at several levels at the whole organism level. The classical actions of PTH on bone, kidney, and gut are dependent upon adequate status of both vitamin D and magnesium. At the same time, high PTH activity increases the requirement for vitamin D and low vitamin D status is commonly associated with both subclinical magnesium deficiency and increased PTH activity.
Keywords
parathyroid hormone; serum calcium; osteomalacia; vitamin D; magnesium; hyperparathyroidism; bone resorption