Okazaki concluded that DNA replication proceeds by a discontinuous mechanism. His data actually suggested that both strands are copied discontinuously. It wasn't until he used a mutant deficient in a particular repair process (uracil excision) that he understood that fragments of one strand produced by this repair had nothing to do with the actual replication process. The small fragments that can be observed during the short radiolabeling periods are called Okazaki fragments in his honor.